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Celiac Disease by Gerard E. Mullin M.D., S. Devi Rampertab M.D., F.A.C.G.

By Gerard E. Mullin M.D., S. Devi Rampertab M.D., F.A.C.G. (auth.), S. Devi Rampertab, Gerard E. Mullin (eds.)

Celiac Disease is an authoritative textual content meant to coach healthcare practitioners, gastroenterologists, physicians and nutritionists in regards to the different manifestations, problems and administration of gluten-sensitive enteropathy. The seventeen-chapter textual content offers the most recent examine on pathophysiology, together with animal versions, immunopathogenesis, genetics and the intestinal microflora. The ebook additionally positive factors chapters on prognosis, tracking, issues and dietary administration, together with treatments at the horizon. The textual content concludes with appendices that offer academic instruments for symptom tracking and nutritional guidance.

Written by way of experts of their respective fields, Celiac Disease is a finished source that gives the healthcare practitioner an evidence-based useful consultant to pathogenesis and treatment of celiac illness.

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Quiz 37, 45. 12. Mustalahti K, Catassi C, Reunanen A, Fabiani E, Heier M, McMillan S, et al. The prevalence of celiac disease in Europe: results of a centralized, international mass screening project. Ann Med. 2010;42(8):587–95. 13. Biagi F, Klersy C, Balduzzi D, Corazza GR. Are we not over-estimating the prevalence of coeliac disease in the general population? Ann Med. 2010;42(8):557–61. 14. Catassi C, Kryszak D, Bhatti B, Sturgeon C, Helzlsouer K, Clipp SL, et al. Natural history of celiac disease autoimmunity in a USA cohort followed since 1974.

High-Risk Groups There are several high-risk groups for CD. One of these is the first-degree relatives [10, 24]. American researchers screened 344 family members of about 100 CD cases, and discovered that more than 10 % of the family members had CD [24]. 5 % were positive for CD. This is likely to constitute a relative risk increase of about ten times [10]. Although it is currently clear that first-degree relatives represent a high-risk group and so they need to be tested for CD at least once in their life, it is not clear whether first-degree relatives found to be negative at a first testing 3 Epidemiology of Celiac Disease 31 need to be further tested in their life.

Dense genotyping identifies and localizes multiple common and rare variant association signals in celiac disease. Nat Genet. 2011;43(12):1193–201. Fasano A, Catassi C. Clinical practice. Celiac disease. N Engl J Med. 2012;367(25): 2419–26. Ludvigsson JF, Leffler DA, Bai JC, Biagi F, Fasano A, Green PH, et al. The Oslo definitions for coeliac disease and related terms. Gut. 2013;62(1):43–52. Sellitto M, Bai G, Serena G, Fricke WF, Sturgeon C, Gajer P, et al. Proof of concept of microbiome-metabolome analysis and delayed gluten exposure on celiac disease autoimmunity in genetically at-risk infants.

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